LaiLab Research

LaiLab logoOur current efforts are investigating the regulatory signalling pathways of muscle atrophy and mitophagy, using different phenotypic models and developing biochemical tools to analyse biological samples.

Tool Development

To explore novel pathways requires novel tools - We seek out the most cutting edge techniques and combine it with classical laboratory methods to have a comprehensive understanding. From CRISPR CAS9 and synthesising recombinant proteins to western blotting and microscopy, our lab utilises the most effective methods for understanding biology.

From Molecules to Life

The only mechanism is the full mechanism - We study cell signalling by understanding protein function. Using biochemical assays to map the signalling cascade we reconciling the gap between in-vitro and in-vivo models.

signalling pathway model image

Current model of signalling pathway determining skeletal muscle mass

Current Research

Understanding the characteristics and function of MuRF1

MuRF1 is an Ubiquitin-E3 ligase implicated in driving muscle atrophy, however there is little understood about its role in muscle protein breakdown. We aim to understand the upstream and downstream signalling pathways, drivers of expression, and potential interacting partners of MuRF1. We also aim to further this research by exploring MuRF2 and MuRF3, also implicated in muscle atrophy.

MuRF structure diagram including molecules

Predicted 3d structure and functional domain locations of MuRF E3 Ligases.3D structure colours denote hydrophobicity: blue=hydrophilic, red=hydrophobic. MFC = MuRF Family Conserved Domain; B-Box = B-Box Zinc Finger Domain; COS = C-terminal Subgroup One Singature; AR = Acidic Region.

Elucidating the mechanisms regulating mitophagy in skeletal muscle.

Mitochondrial turnover is crucial for maintaining cellular homeostasis. The molecular mechanisms governing skeletal muscle mitophagy (Mitochondrial degradation), particularly in response to exercise, are yet to be fully elucidated. We aim to understand the mechanisms that underlie mitophagy in skeletal muscle. This is crucial for developing therapies to combat mitochondrial dysfunction in metabolic disease.

image of two mitophagy proteins with blue and yellow shading

Fluorescence image showing mitophagy in action. Using GFP + mCherry tagged mitochondrial FIS1 protein, mitophagy is observable when GFP is quenched leaving mCherry alone (GFP + mCherry FIS1 construct kindly provided by Dr Ian Ganley, MRC-PPU, University of Dundee).

Resources

Mitochondrial Profiling Centre  

Our facility provides high resolution respirometry platform and expertise. Please contact facilities manager Dr Jonathan Barlow or Dr Yu-Chiang Lai for further details.

Reagents

Our laboratory reagents are available to the scientific community upon request - please see our contacts page.